By John M. Morihisa
Albany clinical university, Albany, manhattan. bargains clinicians and researchers more desirable figuring out of the idea and perform of mind imaging in psychiatry. reports purposes to such problems as melancholy, obsessive-compulsive ailment, and schizophrenia. Softcover. DNLM: psychological problems - prognosis.
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However, the same region of the ACC also showed activity during correct trials in which there was response conflict. This suggests that the ACC does not detect errors per se, which would require some rather complicated comparator process by which it somehow has knowledge of what the correct response should have been as well the actual, incorrect response being executed. Rather, it simply detects conflict between incompatible response tendencies. On this view, errorrelated activity would simply reflect conflict between the incorrect response that has just been executed and an ongoing attempt to execute the correct response as subjects continue to attempt to perform as instructed.
The latter studies typically use single or population neuron recording, focal lesions, or pharmacological manipulations in nonhuman primates or other species performing cognitive tasks. This integration of data from multiple sources has rapidly expanded our understanding of the neural basis of cognition. The unprecedented opportunity for clinical neuroscience is to take this body of knowledge and build upon it, using methodologies such as functional neuroimaging, to develop an understanding of the neural basis of cognitive impairments in mental disorders.
For example, many previous activation studies used complex tasks that could not isolate the neural corCognitive Neuroscience 37 relates of specific cognitive mechanisms. Hence, even though there have now been over 20 years of functional imaging research, and though the data point to alterations in the function of lateral and medial frontal areas associated with executive functions, critical questions remain as to the functional significance of these findings. Is there a selective impairment of DLPFC-related topdown control in schizophrenia, or is the problem an impaired modulation of the DLPFC by the conflict monitoring of the ACC?